By A. James Barkovich
The completely up-to-date Fourth variation of this acclaimed reference describes and illustrates the entire diversity of pediatric problems diagnosable via smooth neuroimaging. This version comprises state of the art details at the use of proton spectroscopy, diffusion imaging, and perfusion imaging in diagnosing metabolic issues, mind tumors, abnormalities of cerebral microstructure, and abnormalities of blood stream. New entities were further to the chapters on metabolic issues, mind accidents, congenital malformations of the mind and cranium, cerebellar problems, mind tumors, phakomatoses, hydrocephalus, and infections. greater than 2,400 photos supplement the textual content. an inventory of problems with corresponding web page numbers permits readers to speedy search for a affliction.
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Extra info for Pediatric Neuroimaging 4th Edition
T1-Weighted Images The changes of white matter maturation are visually detected at different rates and at different times on T1-weighted images than on T2-weighted images. On T1-weighted images, the appearance of the newborn brain is grossly similar to that of T2-weighted images in adults in that white matter has lower signal intensity than gray matter. As white matter matures, its signal intensity increases relative to gray matter. 31 Neonatal posterior fossa structures that exhibit high signal intensity at birth include the medial lemniscus, lateral lemniscus, median longitudinal fasciculus, brachium of the inferior colliculus, and the inferior and superior cerebellar peduncles (Figure 2-7) (37).
Foci of gray matter intensity are seen just anterior to the tips of the frontal horns of the lateral ventricles (arrows in C), representing foci of residual germinal matrix. Hyperintensity on T1-weighted images and hypointensity on T2-weighted images is present in the dorsal brain stem, superior and inferior cerebellar peduncles, far lateral putamen, and ventrolateral thalamic nucleus. The cerebral white matter still appears completely unmyelinated. The cisterna magna and basilar cisterns are relatively large throughout infancy.
It is still very thin, but is barely visible. Note that the cerebellum is still very small at this age. 52 53 Figure 2-16 (continued) E: Normal 1-month-old infant. On this SE 600/20 image, the corpus callosum is isointense with the rest of the brain. The corpus callosum is uniformly thin at this age without the normal bulbous enlargement of the genu and splenium; the genu, body and splenium are all of the same thickness. F: Normal 4-month-old infant. By 3 to 4 months of age, the splenium of the corpus callosum increases in size and begins to show an increased signal intensity as compared to the rest of the brain on SE 600/20 images.