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By Sabatini Rosa

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Extra resources for Adverse Effects of Hormonal Contraceptives: Myth and Reality

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A biochemical evaluation. B. Sex-steroids used in hormonal treatment increase vascular procoagulant activity by inducing thrombin receptor (PAR-1) expression:role of the glucocorticoid receptor. Circulation, 104(23), 282631. , de Moerloose, P. Inherited thrombophilia and arterial diseases. Rev Med Suisse, Feb 7, 3(97), 331-2, 334-5. Review. , Imberti, D. (2006). Semin Thromb Hemost, Oct, 32(7), 651-8. Review. , van der Meer, J. The pathogenesis of venous thromboembolism: evidence for multiple interrelated causes.

Oral contraceptives and venous thrombosis: different sensitivities to activated protein C in women using second-and third-generation oral contraceptives. J Haematol, 97, 233-238. Y. (2004). Impact of progestagens on activated protein C (APC) resistance among users of oral contraceptives. J Thromb Haemost, Sep, 2(9), 1594-600. R. (1997). Population-based study of risk of venous thromboembolism associated with various oral contraceptives. Lancet, 349(9045), 83-8. , Vasilakis, C. (1995). Risk of idiopathic cardiovascular death and nonfatal thrombo embolism in women using oral contraceptives with differing progestagen components.

J. (2005). Probable migraine with visual aura and risk of ischemic stroke:the stroke prevention in young women study. Stroke, 38(9), 2438-45, 330(7482), 63. E. (2005). Association between the current use of low-dose oral contraceptives and cardiovascular arterial disease: a meta-analysis. Clin. Metab, 90(7), 3853-70. , Parma, M. (1998). Minerva Med, 71(30), 2125-9. M. Venous thromboembolism,myocardial infarction,and stroke among transdermal contraceptive system users. Gynecol, 109(2), 339-46. , Crook, D.

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